Laser-Accelerated Inflammation/ Pain Reduction and Healing
by Richard Martin, BS, CLT
 Compromised cells and tissues respond more readily than healthy
cells or tissues to energy transfers that occur between LLLT-emitted photons and the
receptive chromophores found in the various cells and sub-cellular organelles. Cells and
tissues that are ischemic and poorly perfused as a result of inflammation, edema and
injury have been shown to have a significantly higher response to LLLT irradiation than
normal healthy structures. Cell membranes, mitochondria and damaged neurological
structures exhibit less than optimal metabolism and stasis conditions. Multiple studies
have demonstrated that under these compromised conditions, the introduction of energy
transfers and the resultant enhancement of metabolic activity is most pronounced in
biologically challenged components. While it may appear that LLLT is thus selectively
targeting compromised cells, in reality, these cells exhibit a lowered reaction threshold
to the effects of laser light and are more easily triggered to energy transfer responses.
The result is that LLLT has a significant effect on damaged cells and tissues while
normative biological constituents are appreciably less affected.1
The cellular cascade effect — precipitated by the actions of enzymes and having a
significant in the presence of LLLT — has a significant impact on cellular and tissue
function. Since a considerable number of the reactive proteins that respond to laser
stimulation are enzymes, laser light effects are amplified in the stimulation of
beneficial enzymes and depression of deleterious enzymes.
At the cellular level, cytochromes can be defined as electron or proton-transfer
proteins that act as energy producers for human biological functions. Both of the
cytochrome enzymes, Cytochrome c Oxydase and Nitric Oxide Synthase (NOS) have been found
to be particularly reactive to laser photon stimulation. The particular affinity of these
and other photo-reactive enzymes to accelerate their functions in the presence of LLLT
provides critical increases in the molecule ATP and Nitric Oxide (NO) which enhances
cellular metabolism, circulatory improvement and nerve function.
Please refer to the Nov/Dec 2003 issue for the complete text. In the event you need to order a back issue, please click here.
— Nov/Dec 2003
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