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Non-Pharmacologic Treatment of Shingles

by Carolyn McMakin, MA, DC

 In unpublished anecdotal reports of Frequency Specific Microcurrent (FSM) treatments during the last twelve years, one frequency combination has been observed consistently to eliminate the pain and shorten the course of shingles. That frequency combination—230 Hz on one channel and 430 Hz on the second channel—applied simultaneously using 150 microamps alternating DC current having a ramped square wave pulse was ultimately successful on this patient’s pain and lesions.

Case Report
The patient was an 85-year-old male who presented for treatment with Frequency Specific Microcurrent (FSM) of low back pain caused by myofascial trigger points and degenerative disc disease. The patient noted incidentally that he had a rash on the frontal portion of his bald scalp which was diagnosed one week previously by his dermatologist as actinic keratosis. He was applying a topical gel appropriate to that diagnosis and did not request treatment for the rash during this appointment. He returned two days later complaining of increased pain on his scalp in the area of the rash, rated as a 7/10 on a 0-10 VAS scale, and requested that the rash be treated with FSM appropriate for actinic keratosis since treatment had been effective for his low back pain.1

The treatment protocol for actinic keratosis required that microamperage current from a two channel microcurrent device be applied to the skin on his scalp. The current is delivered using graphite conducting gloves with double pin connectors cemented to the back so that one lead from each of two channels can be connected to each glove. One graphite glove, connected to the positive leads from both channels of the microcurrent device, was wrapped in warm moist fabric and placed on the patient’s upper back. The second graphite glove, connected to the negative leads from both channels of the microcurrent device, was wrapped in a warm moist face cloth and placed on the top of the patient’s head so that it covered the area of the rash on the scalp (see Figure 2).

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— May 2010

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